VIP and muscarinic synergistic mucin secretion by salivary mucous cells is mediated by enhanced PKC activity via VIP-induced release of an intracellular Ca2+ pool
Author:
Funder
National Institute of Dental and Craniofacial Research
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s00424-020-02348-7.pdf
Reference114 articles.
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2. Ambudkar I (2018) Calcium signaling defects underlying salivary gland dysfunction. Biochim Biophys Acta Mol Cell Res 1865:1771–1777. https://doi.org/10.1016/j.bbamcr.2018.07.002
3. Ashton N, Evans RL, Elliott AC, Green R, Argent BE (1993) Regulation of fluid secretion and intracellular messengers in isolated rat pancreatic ducts by acetylcholine. J Physiol 471:549–562. https://doi.org/10.1113/jphysiol.1993.sp019915
4. Bhattacharya S, Imbery JF, Ampem PT, Giovannucci DR (2015) Crosstalk between purinergic receptors and canonical signaling pathways in the mouse salivary gland. Cell Calcium 58:589–597. https://doi.org/10.1016/j.ceca.2015.09.006
5. Bou-Hanna C, Berthon B, Combettes L, Claret M, Laboisse CL (1994) Role of calcium in carbachol- and neurotensin-induced mucin exocytosis in a human colonic goblet cell line and cross-talk with the cyclic AMP pathway. Biochem J 299(Pt 2):579–585. https://doi.org/10.1042/bj2990579
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