Human macrophages directly modulate iPSC-derived cardiomyocytes at healthy state and congenital arrhythmia model in vitro
Author:
Funder
Türkiye Bilimsel ve Teknolojik Araştirma Kurumu
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s00424-022-02743-2.pdf
Reference46 articles.
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2. Bolego C, Cignarella A, Staels B, Chinetti-Gbaguidi G (2013) Macrophage function and polarization in cardiovascular disease a role of estrogen signaling? Arterioscler Thromb Vasc Biol 33:1127–1134. https://doi.org/10.1161/ATVBAHA.113.301328
3. C. Wang S, Meng D, Yang H, Wang X, Jia S, Wang Y-F, Wang P (2017) Pathological basis of cardiac arrhythmias: vicious cycle of immune-metabolic dysregulation. Cardiovasc Disord Med 3:1–7. https://doi.org/10.15761/cdm.1000158
4. Cao X, Yakala GK, van den Hil FE, Cochrane A, Mummery CL, Orlova Vv (2019) Differentiation and Functional Comparison of Monocytes and Macrophages from hiPSCs with Peripheral Blood Derivatives. Stem Cell Reports 12:1282–1297. https://doi.org/10.1016/j.stemcr.2019.05.003
5. Chen S, Yang L, Nilsson-Payant B, Han Y, Jaffré F, Zhu J, Wang P, Zhang T, Redmond D, Houghton S, Møller R, Hoagland D, Horiuchi S, Acklin J, Lim J, Bram Y, Richardson C, Chandar V, Borczuk A, Huang Y, Xiang J, Ho D, Schwartz R, tenOever B, Evans T (2020) SARS-CoV-2 Infected Cardiomyocytes Recruit Monocytes by Secreting CCL2. Res Sq. https://doi.org/10.21203/RS.3.RS-94634/V1
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