Slow recovery from fast inactivation of Nav1.3 channels: a common gating mechanism shared in sweet- and sour-sensing cells
Author:
Funder
National Institute of Neurological Disorders and Stroke
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s00424-021-02575-6.pdf
Reference15 articles.
1. Barlow LA (2015) Progress and renewal in gustation: new insights into taste bud development. Development 142:3620–3629. https://doi.org/10.1242/dev.120394
2. Carbone E (2021) Fast inactivation of Nav1.3 channels by FGF14 proteins: an unconventional way to regulate the slow firing of adrenal chromaffin cells. J Gen Physiol 153(5):e202112879. https://doi.org/10.1085/jgp.202112879
3. Cummins TR, Aglieco F, Renganathan M, Herzog RI, Dib-Hajj SD, Waxman SG (2001) Nav1.3 sodium channels: rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons. J Neurosci 21:5952–5961
4. Dover K, Solinas S, D’Angelo E, Goldfarb M (2010) Long-term inactivation particle for voltage-gated sodium channels. J Physiol 588:3695–3711. https://doi.org/10.1113/jphysiol.2010.192559
5. Felts PA, Yokoyama S, Dib-Hajj S, Black JA, Waxman SG (1997) Sodium channel alpha-subunit mRNAs I, II, III, NaG, Na6 and hNE (PN1): different expression patterns in developing rat nervous system. Brain Res Mol Brain Res 45:71–82. https://doi.org/10.1016/s0169-328x(96)00241-0
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