Inhibition of nitric oxide production by aminoguanidine influences the number of Trichinella spiralis parasites in infected “low responders” (C57BL/6) and “high responders” (BALB/c) mice

Author:

Kołodziej-Sobocińska Marta,Dziemian Ewa,Machnicka-Rowińska Barbara

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Insect Science,General Veterinary,General Medicine,Parasitology

Reference11 articles.

1. Bruschi F, Solfanelli S, Binaghi RA (1992) Trichinella spiralis—modifications of the cuticule of the newborn larva during passage through the lung. Exp Parasitol 75(1):1–9

2. Dbrowska J, Walski M, Grytner-Zięcina B, Machnicka-Rowińska B, Dziemian E, Jankowska-Steifer E (2004) Ultrastructural analysis of capsule and nurse cell morphology examined seven months after Trichinella spiralis mouse infection. Wiad Parazytol 50(2):279–284 (in Polish)

3. Espinoza EY, Perez-Arellano JL, Carranza C, Collia F, Muro A (2002) In vivo inhibition of inducible nitric oxide synthase decreases lung injury induced by Toxocara canis in experimentally infected rats. Parasite Immunol 24(11–12):511–520

4. Flanagan DM, Jennings CD, Goes SW, Caywood BE, Gross R, Kaplan AM, Bryson JS (2002) Nitric oxide participates in the intestinal pathology associated with murine syngeneic graft-versus-host disease. J Leukoc Biol 72(4):762–768

5. Helmby H, Grencis RK (2003) IFN-gamma-independent effects of IL-12 during intestinal nematode infection. J Immunol 171(7):3691–3696

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