Decreased hyperammonaemia and orotic aciduria due to inactivation of ornithine aminotransferase in mice with a hereditary abnormal ornithine carbamoyltransferase
Author:
Publisher
Wiley
Subject
Genetics (clinical),Genetics
Link
http://www.springerlink.com/index/pdf/10.1007/BF00712011
Reference20 articles.
1. Bachmann C, Colombo JP (1980) Diagnostic value of orotic acid excretion in heritable disorders of the urea cycle and in hyperammonemia due to organic acidurias.Eur J Pediatr 134: 109?113.
2. Briand P, François B, Rabier D, Cathelineau L (1982) Ornithine transcarbamylase deficiencies in human males; kinetic and immunochemical classification.Biochim Biophys Acta 704: 100?106.
3. Ceriotti G (1974) Ornithine carbamoyltransferase. In Bergmeyer HK, ed.Methods in Enzymatic Analysis, vol. 2. Weinheim: Verlag Chemie, 691?698.
4. Daune G, Gerhart F, Seiler N (1988) 5-Fluoromethylornithine, an irreversible and specific inhibitor ofl-ornithine: 2-oxo-acid aminotransferase.Biochem J 253: 481?488.
5. Daune-Anglard G, Bonaventure N, Seiler N (1993) Some biochemical and pathophysiological aspects of long-term elevation of brain ornithine concentrations.Pharmacol Toxicol 73: 29?34.
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