Expression and regulation of mRNAs for insulin-like growth factor (IGF-I), IGF-binding protein-2, and LH receptor in the process of follicular atresia

Author:

Luo Wenxiang,Zhu Cheng

Publisher

Springer Science and Business Media LLC

Subject

General Agricultural and Biological Sciences,General Environmental Science,General Biochemistry, Genetics and Molecular Biology

Reference15 articles.

1. Manikkam, M., Rajamahendran, R., Progesterone-induced atresia of the proestrous dominant follicles in the bovine ovary: changes in diameter, insulin-like growth factor system, aromatase activity, steroid hormones and apoptotic index, Biol. Reprod., 1997, 57: 580.

2. Tilly, J. L., Ovarian follicullar atresia: a model to study the mechanisms of physiological cell death, Endocr. J., 1993, 1: 67.

3. Hsueh, A. J. W., Billig, H., Tsafriri, A., Ovarian follicle atresia: a hormonally controlled apoptotic process, Endocr. Rev. 1994, 15: 707.

4. Tilly, J. L., Billig, H., Kowalski, K. I. et al., Epidermal growth factor and basic fibroblast growth factor suppress the spontaneous onset of apoptosis in cultured rat granulosa cells and follicles by a tyrosine kinase-dependent mechanism, Mol. Endocrinol., 1992, 6: 1942.

5. Chun, S.Y., Billig, H., Tilly, J.L. et al., Gonadotropin suppression of apoptosis in cultured preovulatory follicles: mediatory role of endogenous insulin-like growth factor I, Endocrinology, 1994, 135: 1845.

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