Polymorphisms of the genes CTLA4, PTPN22, CD40, and PPARG and their roles in Graves’ disease: susceptibility and clinical features
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://link.springer.com/content/pdf/10.1007/s12020-020-02337-x.pdf
Reference44 articles.
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2. Y. Tomer, A. Huber, The etiology of autoimmune thyroid disease: a story of genes and environment. J. Autoimmun. 32, 231–239 (2009). https://doi.org/10.1016/j.jaut.2009.02.007
3. T.H. Brix, K.O. Kyvik, K. Christensen, L. Hegedüs, Evidence for a major role of heredity in Graves’ disease: a population-based study of two Danish twin cohorts. J. Clin. Endocrinol. Metab. 86, 930–934 (2001). https://doi.org/10.1210/jcem.86.2.7242
4. A. Hasham, Y. Tomer, Genetic and epigenetic mechanisms in thyroid autoimmunity. Immunol. Res. 54, 204–213 (2012). https://doi.org/10.1007/s12026-012-8302-x
5. C.E. Rudd, H. Schneider, Unifying concepts in CD28, ICOS and CTLA4 co-receptor signalling. Nat. Rev. Immunol. 3, 544–556 (2003). https://doi.org/10.1038/nri1131
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