Inhibition of Inducible Nitric Oxide Synthase Attenuates Deficits in Synaptic Plasticity and Brain Functions Following Traumatic Brain Injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Clinical Neurology,Neurology
Link
http://link.springer.com/article/10.1007/s12311-018-0934-5/fulltext.html
Reference38 articles.
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2. Thal SC, Neuhaus W. The Blood–brain barrier as a target in traumatic brain injury treatment. Arch Med Res. 2014;45:698–710. https://doi.org/10.1016/j.arcmed.2014.11.006 .
3. Cederberg D, Siesjo P. What has inflammation to do with traumatic brain injury? Child’s Nerv Syst. 2010;26:221–6. https://doi.org/10.1007/s00381-009-1029-x .
4. Zamora R, Vodovotz Y, Billiar TR. Inducible nitric oxide synthase and inflammatory diseases. Mol Med. 2000;6:347–73.
5. Garry PS, Ezra M, Rowland MJ, Westbrook J, Pattinson KTS. The role of the nitric oxide pathway in brain injury and its treatment—from bench to bedside. Exp Neurol. 2015;263:235–43. https://doi.org/10.1016/j.expneurol.2014.10.017 .
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