Potential Benefit of Channel Activators in Loss-of-Function Primary Potassium Channelopathies Causing Heredoataxia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Neurology
Link
https://link.springer.com/content/pdf/10.1007/s12311-023-01584-8.pdf
Reference38 articles.
1. Huang H, Shakkottai VG. Targeting ion channels and Purkinje neuron intrinsic membrane excitability as a therapeutic strategy for cerebellar ataxia. Life. 2023;13:1350.
2. Srinivasan SR, Huang H, Chang WC, Nasburg JA, Nguyen HM, Strassmaier T, et al. Discovery of novel activators of large-conductance calcium-activated potassium channels for the treatment of cerebellar ataxia. Mol Pharmacol. 2022;102:438–49.
3. Womack MD, Khodakhah K. Somatic and dendritic small-conductance calcium-activated potassium channels regulate the output of cerebellar Purkinje neurons. J Neurosci. 2003;23:2600–7.
4. Pulst SM, Otis TS. Repolarization matters: mutations in the Kv4.3 potassium channel cause SCA19/22. Ann Neurol. 2012;72:829–31.
5. Köhling R, Wolfart J. Potassium channels in epilepsy. Cold Spring Harb Perspect Med. 2016;6: a022871.
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