Endoplasmic Reticulum Stress and Autophagy in the Pathogenesis of Non-alcoholic Fatty Liver Disease (NAFLD): Current Evidence and Perspectives
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s13679-021-00431-3.pdf
Reference234 articles.
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3. Fouad Y, Waked I, Bollipo S, Gomaa A, Ajlouni Y, Attia D. What's in a name? Renaming 'NAFLD' to 'MAFLD'. Liver Int. 2020;40(6):1254–61. https://doi.org/10.1111/liv.14478.
4. Hardy T, Oakley F, Anstee QM, Day CP. Nonalcoholic Fatty Liver Disease: Pathogenesis and Disease Spectrum. Annu Rev Pathol. 2016;11:451–96. https://doi.org/10.1146/annurev-pathol-012615-044224.
5. • Rutkowski DT. Liver function and dysfunction - a unique window into the physiological reach of ER stress and the unfolded protein response. FEBS J. 2019;286(2):356–78. https://doi.org/10.1111/febs.14389The liver due to its unique properties as a highly secretory tissue which regulates peripheral metabolism and can proliferate upon damage underlined the fact that the UPR extends beyond the improvement of ER protein folding to control of metabolism and inflammation in terms of regulating the cycles of cell death and immune cell recruitment.
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