Capacitative Ca2+ entry contributes to the Ca2+ influx induced by thyrotropin-releasing hormone (TRH) in GH3 pituitary cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/BF01837406.pdf
Reference34 articles.
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2. Albert PR, Tashjian AH Jr (1984) Relationship of thyrotropin-releasing hormone-induced spike and plateau phases in cytosolic free Ca2+ concentrations to hormone secretion. Selective blockade using iomomycin and nifedipine J Biol Chem 259:15350–15363
3. Alvarez J, Montero M, García-Sancho J (1992) Cytochrome P450 may regulate plasma membrane Ca2+ permeability according to the filling state of the intracellular Ca2+ stores. FASEB J 6:786–792
4. Barros F, Villalobos C, García-Sancho J, Camino D, Peña P (1994) The role of the inwardly rectifying K+ current in resting potential and thyrotropin-releasing hormone-induced changes in cell excitability of GH3 rat anterior pituitary cells. Pflügers Arch 426:221–230
5. Bjoro T, Sand O, Ostberg BC, Gordeladze JO, Torjesen P, Gautvik KM, Haug E (1990) The mechanisms by which vasoactive intestinal peptide (VIP) and thyrotropin-releasing hormone (TRH) stimulate prolactin release from the pituitary cells. Biosci Rep 10:189–199
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