Appearance of the slow Ca conductance in myotubes from mutant mice with ?muscular dysgenesis?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/BF00585050.pdf
Reference26 articles.
1. Banker BQ (1977) Muscular dysgenesis in the mouse (mdg/mdg). I. Ultrastructural study of skeletal and cardiac muscle. J Neuropathol Exp Neurol 36:100?127
2. Beam KG, Knudson CM (1988) Calcium currents in embryonic and neonatal mammalian skeletal muscle. J Gen Physiol 91:780?798
3. Beam KG, Knudson CM, Powell JA (1986) A lethal mutation in mice eliminates the slow calcium current in skeletal muscle cells. Nature 320:168?170
4. Blau HM, Pavlath GK, Hardeman EC, Chiu C-P, Silberstein L, Webster SG, Miller SC, Webster C (1985) Plasticity of the differentiated state. Science 230:758?765
5. Bournaud R, Mallart A (1987) An electrophysiological study of skeletal muscle fibers in the ?muscular dysgenesis? mutation of the mouse. Pflügers Arch 409:468?476
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1. Molecular Origin of the L-Type Ca2+ Current of Skeletal Muscle Myotubes Selectively Deficient in Dihydropyridine Receptor β1a Subunit;Biophysical Journal;1998-07
2. Involvement of the Dihydropyridine Receptor and Internal Ca2+Stores in Myoblast Fusion;Experimental Cell Research;1996-03
3. CA2+ Entry through Acetylcholine Receptor Channel in Dysgenic Myotubes;Archives of Physiology and Biochemistry;1996-01
4. Myotube driven myogenic recruitment of cells during in vitro myogenesis;Developmental Dynamics;1995-02
5. Effect of SR33557 on Intramembrane Charge Movement in Normal and‘Muscular Dysgenesis’Mouse Skeletal Muscle Cells;European Journal of Neuroscience;1995-01
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