Sodium-dependent Potassium Channels in Leech P Neurons
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology,Biophysics
Link
http://link.springer.com/content/pdf/10.1007/s00232-005-0816-x.pdf
Reference42 articles.
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2. Baylor D.A., Nicholls J.G. 1969. After-effects of nerve impulses on signalling in the central nervous system of the leech. J. Physiol. 203:571–589
3. Bhattacharjee A., Joiner W.J., Wu M., Yang Y., Sigworth F.J., Kaczmarek L.K. 2003. Slick (Slo2.1), a rapidly-gating sodium-activated potassium channel inhibited by ATP. J. Neurosci. 23:11681–11691
4. Bhattacharjee A., von Hehn C.A., Mei X., Kaczmarek L.K. 2005. Localization of the Na(+)-activated K(+) channel Slick in the rat central nervous system. J. Comp. Neurol. 484:80–92
5. Bhattacharjee A., Kaczmarek L.K. 2005. For K+ channels, Na+ is the new Ca2+. Trends Neurosci. 28:422–428
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1. Non-synaptic Plasticity in Leech Touch Cells;Frontiers in Physiology;2019-11-27
2. Choline and NMDG directly reduce outward currents: reduced outward current when these substances replace Na+ is alone not evidence of Na+-activated K+ currents;Journal of Neurophysiology;2018-12-01
3. An annotated CNS transcriptome of the medicinal leech, Hirudo verbana: De novo sequencing to characterize genes associated with nervous system activity;PLOS ONE;2018-07-20
4. Slack, Slick, and Sodium-Activated Potassium Channels;ISRN Neuroscience;2013-05-13
5. Functional properties and cell type specific distribution ofIh channels in leech neurons;Journal of Experimental Biology;2012-01-15
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