Both Early and Late Stages of Hepatocarcinogenesis Are Enhanced in Cx32 Dominant Negative Mutant Transgenic Rats with Disrupted Gap Junctional Intercellular Communication-Reference-Cited by-同舟云学术

Both Early and Late Stages of Hepatocarcinogenesis Are Enhanced in Cx32 Dominant Negative Mutant Transgenic Rats with Disrupted Gap Junctional Intercellular Communication

Published:2007-11-03 Issue:1-3 Volume:218 Page:101-106
ISSN:0022-2631
Container-title:Journal of Membrane Biology
language:en
Short-container-title:J Membrane Biol

Author:

Hokaiwado Naomi,Asamoto Makoto,Futakuchi Mitsuru,Ogawa Kumiko,Takahashi Satoru,Shirai Tomoyuki

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Physiology,Biophysics

Link

http://link.springer.com/content/pdf/10.1007/s00232-007-9053-9.pdf

Reference22 articles.

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2. Dagli ML, Yamasaki H, Krutovskikh V, Omori Y (2004) Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver. Carcinogenesis 25:483–492

3. Evert M, Ott T, Temme A, Willecke K, Dombrowski F (2002) Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice. Carcinogenesis 23:697–703

4. Futakuchi M, Hirose M, Ogiso T, Kato K, Sano M, Ogawa K, Shirai T (1999) Establishment of an in vivo highly metastatic rat hepatocellular carcinoma model. Jpn J Cancer Res 90:1196–1202

5. Hokaiwado N, Asamoto M, Ogawa K, Shirai T (2005) Transgenic disruption of gap junctional intercellular communication enhances early but not late stage hepatocarcinogenesis in the rat. Toxicol Pathol 33:695–701

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