Mechanisms involved in platelet hyperactivation and platelet-endothelium interrelationships in diabetes mellitus
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11892-002-0020-7.pdf
Reference37 articles.
1. Vinik AI, Erbas T, Park TS, et al.: Platelet dysfunction in type 2 diabetes. Diabetes Care 2001, 24:1476–1485.
2. Carr ME: Diabetes mellitus: a hypercoagulable state. J Diabetes Complications 2001, 15:44–54.
3. Srivastava K, Dash D: Changes in membrane microenvironment and signal transduction in platelets from NIDDM patients— a pilot study. Clin Chim Acta 2002, 317:213–220. Demonstrates that resting platelets from type 2 diabetic patients present protein tyrosine phosphorylation, a marker of activation, similar to that of agonist-stimulated platelets from nondiabetic controls.
4. Li Y, Woo V, Bose R: Platelet hyperactivity and abnormal Ca2+ homeostasis in diabetes mellitus. Am J Physiol Heart Circ Physiol 2001, 280:H1480–1489. Shows that in platelets from diabetic patients the sodium/calcium exchanger works in reverse mode, transporting calcium into the cells and not removing it from the cells, a mechanism involved in the increase of platelet calcium concentrations.
5. Wieclawska B, Rozalski M, Trojanowski Z, Watala C: Modulators of intraplatelet calcium concentration affect the binding of thrombospondin to blood platelets in healthy donors and patients with type 2 diabetes. Eur J Haematol 2001, 66:396–403.
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