Role for SUMOylation in disturbed flow-induced atherosclerotic plaque formation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Biomedical Engineering
Link
http://link.springer.com/article/10.1007/s13534-015-0199-6/fulltext.html
Reference83 articles.
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2. Heo KS, Fujiwara K, Abe J. Disturbed-flow-mediated vascular reactive oxygen species induce endothelial dysfunction. Circ J. 2011; 75(12):2722–30.
3. Heo KS, Chang E, Le NT, Cushman H, Yeh ET, Fujiwara K, Abe J. De-SUMOylation enzyme of sentrin/SUMO-specific protease 2 regulates disturbed flow-induced SUMOylation of ERK5 and p53 that leads to endothelial dysfunction and atherosclerosis. Circ Res. 2013; 112(6):911–23.
4. Heo KS, Le NT, Cushman HJ, Giancursio CJ, Chang E, Woo CH, Sullivan MA, Taunton J, Yeh ET, Fujiwara K, Abe J. Disturbed flow-activated p90RSK kinase accelerates atherosclerosis by inhibiting SENP2 function. J Clin Invest. 2015; 125(3): 1299–310.
5. Urbich C, Stein M, Reisinger K, Kaufmann R, Dimmeler S, Gille J. Fluid shear stress-induced transcriptional activation of the vascular endothelial growth factor receptor-2 gene requires Sp1-dependent DNA binding. FEBS Lett. 2003; 535(1–3):87–93.
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1. The Role of Protein SUMOylation in the Pathogenesis of Atherosclerosis;Journal of Clinical Medicine;2019-11-02
2. Special issue on mechanobiology and diseases;Biomedical Engineering Letters;2015-09
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