Expression of human O6-methyl guanine methyl transferase (MGMT) in post replication repair (PRR) deficient CHO-UV-1 cells: Compensation for hypersensitivity to methylating and ethylating agents but not to mitomycin C-Reference-Cited by-同舟云学术

Expression of human O6-methyl guanine methyl transferase (MGMT) in post replication repair (PRR) deficient CHO-UV-1 cells: Compensation for hypersensitivity to methylating and ethylating agents but not to mitomycin C

Published:1997-01 Issue:1 Volume:23 Page:9-17
ISSN:0740-7750
Container-title:Somatic Cell and Molecular Genetics
language:en
Short-container-title:Somat Cell Mol Genet

Author:

Gustafson Daniel L.,Trotter Bonnie K.,Snead Denise,Waldren Charles A.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Genetics,Genetics,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/BF02679951.pdf

Reference26 articles.

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2. Stamato, T.D., Hinkle, L., Collins, A.R.S., and Waldren, C.A. (1981).Somatic Cell Genet. 7:307–320.

3. Stamato, T.D., Waldren, C.A. (1977).Somatic Cell Genet. 3:431–440.

4. Hoy, C.A., and Thompson, L.H., Salazar, E.P., and Stewart, S.A. (1985).Somat. Cell Molec. Genet. 11:523–532.

5. Waldren, C., Snead, D., and Stamato, T. (1983). Restoration of normal resistance to killing and of post-replication recovery (PRR) in CHO-UV-1 cells by transformation with hamster or human DNA. InCellular Responses to DNA Damage. (eds.) E.C. Friedberg, B.A. Bridges. (Alan Liss, Inc., New York), pp. 637–646.

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1. In vivo kinetics of micronuclei induction by bifunctional alkylating antineoplastics;Mutagenesis;2004-05-01

2. Arsenic toxicity is enzyme specific and its affects on ligation are not caused by the direct inhibition of DNA repair enzymes;Mutation Research/DNA Repair;1998-09