Deficits in Responding to Brief Noise Offsets in Kcna1 −/− Mice Reveal a Contribution of This Gene to Precise Temporal Processing Seen Previously Only for Stimulus Onsets
Author:
Publisher
Springer Science and Business Media LLC
Subject
Sensory Systems,Otorhinolaryngology
Link
http://link.springer.com/content/pdf/10.1007/s10162-011-0312-1.pdf
Reference37 articles.
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2. Allen PD, Virag TM, Ison JR (2002) Humans detect gaps in broadband noise according to effective quiet time without additional cues from abrupt envelope changes. J Acoust Soc Am 112:2967–2974
3. Allen PD, Schmuck N, Ison JR, Walton JP (2008) Kv1.1 channel sub-units are not necessary for high temporal acuity in behavioral and electrophysiological gap detection. Hear Res 246:52–58
4. Bal R, Oertel D (2001) Potassium currents in octopus cells of the mammalian cochlear nucleus. J Neurophys 86:2299–2311
5. Békésy G (1933/1960) Experiments in hearing. McGraw-Hill, New York
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2. Sound Localization in Preweanling Mice Was More Severely Affected by Deleting the Kcna1 Gene Compared to Deleting Kcna2, and a Curious Inverted-U Course of Development That Appeared to Exceed Adult Performance Was Observed in All Groups;Journal of the Association for Research in Otolaryngology;2019-08-13
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5. Deficit in acoustic signal‐in‐noise detection in glycine receptor α3 subunit knockout mice;European Journal of Neuroscience;2017-01-18
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