Distorted frequency of dendritic cells and their associated stimulatory and inhibitory markers augment the pathogenesis of pemphigus vulgaris
Author:
Funder
All-India Institute of Medical Sciences
Indian Council of Medical Research
Publisher
Springer Science and Business Media LLC
Subject
Immunology
Link
http://link.springer.com/content/pdf/10.1007/s12026-020-09166-0.pdf
Reference44 articles.
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3. Bettelli E, Carrier Y, Gao W, Korn T, Strom TB, Oukka M, et al. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells. Nature. 2006;441:235–8. https://doi.org/10.1038/nature04753.
4. Asothai R, Anand V, Das D, Antil PS, Khandpur S, Sharma V, et al. Distinctive Treg associated CCR4-CCL22 expression profile with altered frequency of Th17/Treg cell in the immunopathogenesis of pemphigus vulgaris. Immunobiology. 2015;220:1129–35. https://doi.org/10.1016/J.IMBIO.2015.06.008.
5. Piccioli D, Tavarini S, Borgogni E, Steri V, Nuti S, Sammicheli C, et al. Functional specialization of human circulating CD16 and CD1c myeloid dendritic-cell subsets. Blood. 2007;109:5371–9. https://doi.org/10.1182/blood-2006-08-038422.
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