Over-expression of LRIG1 suppresses biological function of pituitary adenoma via attenuation of PI3K/AKT and Ras/Raf/ERK pathways in vivo and in vitro-Reference-Cited by-同舟云学术

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Over-expression of LRIG1 suppresses biological function of pituitary adenoma via attenuation of PI3K/AKT and Ras/Raf/ERK pathways in vivo and in vitro

Published:2016-07-28 Issue:4 Volume:36 Page:558-563
ISSN:1672-0733
Container-title:Journal of Huazhong University of Science and Technology [Medical Sciences]
language:en
Short-container-title:J. Huazhong Univ. Sci. Technol. [Med. Sci.]

Author:

Cheng Shi-qi,Fan Heng-yi,Xu Xin,Gao Wei-wei,Lv Shi-gang,Ye Min-hua,Wu Miao-jing,Shen Xiao-li,Cheng Zu-jue,Zhu Xin-gen,Zhang Yan

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Biomedical Engineering,Biomaterials,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11596-016-1625-4.pdf

Reference34 articles.

1. Di Ieva A, Rotondo F, Syro L V, et al. Aggressive pituitary adenomas—diagnosis and emerging treatments. Nat Rev Endocrinol, 2014,10(7):423–435

2. Melmed S. Mechanisms for pituitary tumorigenesis: the plastic pituitary. J Clin Invest, 2003,112(11):1603–1618

3. Gillam MP, Molitch ME, Lombardi G, et al. Advances in the treatment of prolactinomas. Endocr Rev, 2006,27(5):485–534

4. Newell-Price J, Bertagna X, Grossman A B, et al. Cushing's syndrome. Lancet, 2006,367(9522):1605–1617

5. Baker NE, Yu SY. The EGF receptor defines domains of cell cycle progression and survival to regulate cell number in the developing Drosophila eye. Cell, 2001,104(5):699–708

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