LRP6 Bidirectionally Regulates Insulin Sensitivity through Insulin Receptor and S6K Signaling in Rats with CG-IUGR-Reference-Cited by-同舟云学术

LRP6 Bidirectionally Regulates Insulin Sensitivity through Insulin Receptor and S6K Signaling in Rats with CG-IUGR

Published:2023-03-13 Issue:2 Volume:43 Page:274-283
ISSN:2096-5230
Container-title:Current Medical Science
language:en
Short-container-title:CURR MED SCI

Author:

Xie Xue-mei,Cao Qiu-li,Sun Yu-jie,Zhang Jie,Liu Kai-li,Qin Ying-fen,Long Wen-jun,Luo Zuo-jie,Li Xiao-wei,Liang Xing-huan,Yuan Guan-dou,Luo Xiao-ping,Xuan Xiu-ping

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Biochemistry

Link

https://link.springer.com/content/pdf/10.1007/s11596-022-2683-4.pdf

Reference62 articles.

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2. Xie X, Lin T, Zhang M, et al. IUGR with infantile overnutrition programs an insulin-resistant phenotype through DNA methylation of peroxisome proliferator-activated receptor-γ coactivator-1α in rats. Pediatr Res, 2015,77(5):625–632

3. Casey PH, Whiteside-mansell L, Barrett K, et al. Impact of prenatal and/or postnatal growth problems in low birth weight preterm infants on school-age outcomes: an 8-year longitudinal evaluation. Pediatrics, 2006,118(3):1078–1086

4. Ong KK. Catch-up growth in small for gestational age babies: good or bad? Curr Opin Endocrinol Diabetes Obes, 2007,14(1):30–34

5. Hermann GM, Miller RL, Erkonen GE, et al. Neonatal catch up growth increases diabetes susceptibility but improves behavioral and cardiovascular outcomes of low birth weight male mice. Pediatr Res, 2009,66(1):53–58

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