Skeletal Muscle Function, Oxygenation and Biochemistry in an Endotoxemic Model of Sirs

Author:

Iannoli Ellen Dailor,Gayeski Thomas E. J.

Publisher

Springer US

Reference25 articles.

1. R.S. Cotran, V. Kumar, and S.L. Robbins, Robbins Pathologic Basis of Disease, W.B.Saunders Company, Philadelphia, (1989).

2. J. Olgin, R.J. Connett, and B. Chance, Mitochondrial redox changes during rest-work transition in dog gracilis muscle. Advances In Experimental Medicine & Biology 200:545–54 (1986).

3. R.J. Connett, T.E. Gayeski, and C.R. Honig, Lactate accumulation in fully aerobic, working, dog gracilis muscle, American Journal of Physiology 246:H120–H128(1984).

4. T.E. Gayeski and C.R. Honig, 02 gradients from sarcolemma to cell interior in red muscle at maximal VO2, American Journal of Physiology 251:H789–H799 (1986).

5. T.E. Gayeski, R.J. Connett, and C.R. Honig, Minimum intracellular PO2 for maximum cytochrome turnover in red muscle in situ, American Journal of Physiology 252:H906–l5 (1987).

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