Endogenous Opioid Peptides, Glucocorticoids, and Shock: Specificity, Site, and Mechanism of Action-Reference-Cited by-同舟云学术

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Endogenous Opioid Peptides, Glucocorticoids, and Shock: Specificity, Site, and Mechanism of Action

Published:1991 Issue: Volume: Page:137-152
ISSN:
Container-title:Central Neural Mechanisms in Cardiovascular Regulation
language:
Short-container-title:

Author:

van Giersbergen Paul L. M.,van den Berg Maria H.,Eijgelshoven Marcel H. J.,Put Joke Cox-van,van den Berg Desirée T. W. M.,de Kloet E. Ronald,de Jong Wybren

Publisher

Birkhäuser Boston

Link

http://link.springer.com/content/pdf/10.1007/978-1-4615-9834-3_11

Reference77 articles.

1. Amir S (1984a): Beneficial effects of i.E.V. naloxone in anaphylactic shock is mediated through peripheral beta-adrenoceptive mechanisms. Brain Res 290: 191–194.

2. Amir S (1984b): Naloxone improves, and morphine exacerbates, experimental shock induced by release of histamine by compound 48/80. Brain Res 297: 187–190.

3. Amir S, Ree JM (1985): Beneficial effect of y-endorphin-type peptides in anaphylactic shock. Brain Res 329: 329–333.

4. Atweh SF, Kuhar MJ (1977): Autoradiographic localization of opiate receptors in rat brain. I. Spinal cord and lower medulla. Brain Res 124: 53–67.

5. Bertolini A, Ferrari W, Guarini S (1989): The adrenocorticotropic hormone ( ACTH)-induced reversal of hemorrhagic shock. Resuscitation 18: 253–267.

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1. Cyclo(Gly-Gln) inhibits the cardiorespiratory depression produced by β-endorphin and morphine;Brain Research;1997-01

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