Author:
Hoehner Jeff,Wechsler Andrew S.,Abd-Elfattah Anwar-Saad A.
Reference22 articles.
1. Abd-Elfattah AS, Jessen ME, Lekven J, Doherty NE III, Brunsting LA, Wechsler AS: Myocardial reperfusion injury: Role of myocardial hypoxanthine and xanthine on “free-radical-mediated reperfusion injury. Circulation 1988;78(Suppl III):224–235.
2. Abd-Elfattah AS, Jessen ME, Hanan SA, Tuchy G, Wechsler AS: Is adenosine-5′-triphosphate derangement or free-radical-mediated injury the major cause of ventricular dysfunction during reperfusion? Role of adenine nucleoside transport in myocardial reperfusion injury. Circulation 1990;82(Suppl IV):IV–341–350.
3. Abd-Elfattah AS, Wechsler AS: Separation between ischemic and reperfusion injury by site specific entrapment of endogenous adenosine and inosine using NBMPR and EHNA J Cardd Surg 1994;9[Suppl]:387–396.
4. Abd-Elfattah AS, Messier RH Jr., Domkowski MS, Jones JL, Aly HM, Crescenzo DG, Wallace RB, Hopkins RA Inhibition of adenosine deaminase and nucleoside transport: Utility in a model of homograft cardiac valve preimplantation processing. J Thorac Cardiovasc Surg 1993;105:1095–1105.
5. Zughaib ME, Abd-Elfattah AS, Jeroudi MO, Sun J-Z, Sekili S, Tang X-L, Bolli R: Augmentation of endogenous adenosine attenuates myocardial “stunning” independently of coronary flow or hemodynqmic effects. Circulation 1993;88:[part 1]:2359–2369.