Prolonged Src Kinase Activation, a Mechanism to Turn Transient, Sublytic Complement Activation into a Sustained Pathological Condition in Retinal Pigment Epithelium Cells-Reference-Cited by-同舟云学术

Prolonged Src Kinase Activation, a Mechanism to Turn Transient, Sublytic Complement Activation into a Sustained Pathological Condition in Retinal Pigment Epithelium Cells

Published:2014 Issue: Volume: Page:221-227
ISSN:0065-2598
Container-title:Retinal Degenerative Diseases
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Author:

Rohrer Bärbel,Kunchithapautham Kannan,Genewsky Andreas,Strauß Olaf

Publisher

Springer New York

Link

http://link.springer.com/content/pdf/10.1007/978-1-4614-3209-8_29

Reference20 articles.

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2. Nowak JZ (2006) Age-related macular degeneration (AMD): pathogenesis and therapy. Pharmacol Rep 58:353–363

3. Snodderly DM (1995) Evidence for protection against age-related macular degeneration by carotenoids and antioxidant vitamins. Am J Clin Nutr 62(6 Suppl):1448S–1461S

4. Charbel Issa P, Chong NV, Scholl HP (2011) The significance of the complement system for the pathogenesis of age-related macular degeneration-current evidence and translation into clinical application. Graefes Arch Clin Exp Ophthalmol 249:163–174

5. Muller-Eberhard HJ (1988) Molecular organization and function of the complement system. Annu Rev Biochem 57:321–347

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