Transcriptional Stress by Camptothecin: Mechanisms and Implications for the Drug Antitumor Activity
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Publisher
Springer New York
Link
http://link.springer.com/content/pdf/10.1007/978-1-4614-0323-4_14
Reference83 articles.
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2. Andersen, F. F., T. O. Tange, et al. (2002). “The RNA splicing factor ASF/SF2 inhibits human topoisomerase I mediated DNA relaxation.” J Mol Biol 322(4): 677–86.
3. Anderson, V. E. and N. Osheroff (2001). “Type II topoisomerases as targets for quinolone antibacterials: turning Dr. Jekyll into Mr. Hyde.” Curr Pharm Des 7(5): 337–53.
4. Baranello, L., D. Bertozzi, et al. (2010). “DNA topoisomerase I inhibition by camptothecin induces escape of RNA polymerase II from promoter-proximal pause site, antisense transcription and histone acetylation at the human HIF-1alpha gene locus.” Nucleic Acids Res 38(1): 159–71.
5. Barboric, M., T. Lenasi, et al. (2009). “7SK snRNP/P-TEFb couples transcription elongation with alternative splicing and is essential for vertebrate development.” Proc Natl Acad Sci USA 106(19): 7798–803.
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