1. Benjamin, W. H., Jr., Hall, P. and Briles, D. E., Salmonella typhimurium locus mviA regulates virulence in Ity s but not Ity r mice: functional mviA results in avirulence; mutant (nonfunctional) mviA results in virulence., J. Exp. Med. 174:1073 (1991).
2. Benjamin, W. H., Jr., Hall, P., Roberts, S. J. and Briles, D. E., The primary effect of the Ity locus is on the growth rate of Salmonella typhimurium that are relatively protected from killing, J. Immunol. 144:3143 (1990).
3. Benjamin, W. H., Jr., Turnbough, C. L., Jr., Goguen, J. D., Posey, B. S. and Briles, D. E., Genetic mapping of novel virulence determents of Salmonella typhimurium to the region between trpD and supD., Microbial Pathogenesis. 1:115 (1986).
4. Benjamin, W. H., Jr., Turnbough, C. L., Jr., Posey, B. S. and Briles., D. E., The ability of Salmonella typhimurium to produce the siderophore enterobactin is not a virulence factor in mouse typhoid., Infect. Immun. 50:392 (1985).
5. Bradley, D. J., Taylor, B. A., Blackwell, J., Evans, E. P. and Freeman, J., Regulation of Leishmania populations within the host III Mapping of the locus controlling susceptibility to visceral leishmaniasis in the mouse., Clin. Exp. Immunol. 37:7 (1979).