Author:
Josse Denis,Masson Patrick,Bartels Cynthia,Lockridge Oksana
Reference58 articles.
1. Adkins S, Gan KN, Mody M, La Du BN (1993) Molecular basis for the polymorphic forms of human serum paraoxonase/arylesterase: glutamine or arginine at position 191, for the respective A or B allozymes. Am Hum Genet 52: 598–608
2. Aviram M (1999) Does paraoxonase play a role in susceptibility to cardiovascular disease? Mol. Med Today 5: 381–386
3. Aviram M, Billecke S, Sorenson R, Bisgaier C, Newton R, Rosenblat M, Erogul J, Hsu C, Dunlop C, La Du B (1998) Paraoxonase active site required for protection against LDL oxidation involves its free sulfhydryl group and is different from that required for its arylesterase/paraoxonase activities: selective action of human paraoxonase allozymes Q and R. Arterioscler Thromb Vasc Biol 18: 1617–1624
4. Aviram M, Hardak E, Vaya J, Mahmood S, Milo S, Hoffman A, Billicke S, Draganov D, Rosenblat M (2000) Human serum paraoxonases (PON1) Q and R selectively decrease lipid peroxides in human coronary and carotid atherosclerotic lesions: PON1 esterase and peroxidase-like activities. Circulation, 101, 2510–2517.
5. Banzon JA, Kuo JM, Fischer DR, Stang PJ, Raushel FM (1995) Histidine-254 is essential for the inactivation of Phosphodiesterase with the alkynyl phosphate esters and diethyl pyrocarbonate. Biochemistry 34: 750–754
Cited by
3 articles.
订阅此论文施引文献
订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献