Kv1.1
Author:
Publisher
Springer New York
Link
http://link.springer.com/content/pdf/10.1007/978-1-4614-6438-9_101677-1
Reference40 articles.
1. Bagchi B, Al-Sabi A, Kaza S, Scholz D, O’Leary VB, Dolly JO, et al. Disruption of myelin leads to ectopic expression of K(V)1.1 channels with abnormal conductivity of optic nerve axons in a cuprizone-induced model of demyelination. PLoS One. 2014;9:e87736. doi:10.1371/journal.pone.0087736.
2. Brew HM, Gittelman JX, Silverstein RS, Hanks TD, Demas VP, Robinson LC, et al. Seizures and reduced life span in mice lacking the potassium channel subunit Kv1.2, but hypoexcitability and enlarged Kv1 currents in auditory neurons. J Neurophysiol. 2007;98:1501–25. doi:10.1152/jn.00640.2006.
3. Browne DL, Gancher ST, Nutt JG, Brunt ER, Smith EA, Kramer P, et al. Episodic ataxia/myokymia syndrome is associated with point mutations in the human potassium channel gene, KCNA1. Nat Genet. 1994;8:136–40. doi:10.1038/ng1094-136.
4. Covarrubias M, Wei AA, Salkoff L. Shaker, Shal, Shab, and Shaw express independent K+ current systems. Neuron. 1991;7:763–73.
5. Dodson PD, Barker MC, Forsythe ID. Two heteromeric Kv1 potassium channels differentially regulate action potential firing. J Neurosci. 2002;22:6953–61.
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