Pathogenesis of Perivenous and Demyelinating Encephalomyelitis and Its Relevance for Multiple Sclerosis Research

Author:

Wisniewski H. M.,Lassmann Hans,Schuller-Levis G.,Mehta P. D.,Madrid R. E.

Publisher

Springer US

Reference90 articles.

1. Alvord, E. C., 1983, Historical perspective and challenge: How good a model of Multiple Sclerosis is experimental allergic encephalomyelitis today? Abstract from the Satellite Meeting of the International Society of Neurochemistry, Seattle, Washington.

2. Antel, J. P., Arnasan, B. G. W., and Medof, M.E., 1979, Suppressor cell function in multiple sclerosis: correlation with clinical disease activity, Ann.Neurol, 5: 338.

3. Benacerraf, B., and McDevitt, H., 1972, Histompatibility-linked immune response genes, Science 175: 273.

4. Ben-Nun, A., and Lando, Z., 1983, Detection of autoimmune cells proliferating to myelin basic protein and selection of T cell lines that mediate experimental autoimmune encephalomyelitis (EAE) in mice, J.Immunol, 130: 1205.

5. Bloom, B., 1980, Immunological changes in multiple sclerosis, Nature (London) 287: 275.

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