Hepatocarcinogenesis by Non-Genotoxic Compounds

Author:

Schulte-Hermann R.,Parzefall W.,Bursch W.,Timmermann-Trosiener I.

Publisher

Springer US

Reference29 articles.

1. Argyris, T.S., Magnus, D.R., 1968, The stimulation of liver growth and demethylase activity following phenobarbital treatment. Dev. Biol. 17: 187.

2. Bieri, F., Bentley, P., Waechter, F., Stäubli, W., 1984, Use of primary cultures of adult rat hepatocytes to investigate mechanisms of action of nafenopin, a hepatocarcinogenic peroxisome proliferator. Carcinogenesis 5: 1033.

3. Bursch, W., Dusterberg, B., Schulte-Hermann, R., 1986, Growth, regression and cell death in rat liver as related to tissue levels of the hepatomitogen cyproterone acetate. Arch. Toxicol. 59: 221.

4. Bursch, W., Lauer, B,. Timmermann-Trosiener, I., Barthel, G., Schuppler, J., Schulte-Hermann, R., 1984, Controlled death ( Apoptosis) of normal and putative preneoplastic cells in rat liver following withdrawal of tumor promoters. Carcinogenesis 5: 453.

5. Bursch, W., Liehr, J., Sirbasku, D., Schulte-Hermann, R., 1988, Role of cell death for growth and regression of hormone-dependent H-301 hamster kidney tumors. in: “Chemical Carcinogenesis: Models and Mechanisms”, F. Feo, P. Pani, A. Columbano, R. Garcea (eds.), Plenum press, in press.

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