Sodium Overload and Mitochondrial Damage in Ischemic/Reperfused Heart

Author:

Takeo Satoshi,Iwai Takeshi,Tanonaka Kouichi

Publisher

Springer US

Reference42 articles.

1. Pierce GN, Czubryt MP. 1995. The contribution of ionic imbalance to ischemia/reperfusion-induced injury. J Mol Cell Cardiol 27:53–63.

2. Pike MM, Luo CS, Clark MD, Kirk KA, Kitakaze M, Madden MC, Cragoe EJ Jr, Pohost GM. 1993. NMR measurements of Na+ and cellular energy in ischemic rat heart: role of Na+-H+ exchange. Am J Physiol 265:H2017–H2026.

3. Hendrix M, Mubagwa K, Verdonck F, Overloop K, Van Heck P, Vanstapel F, Van Lommel A, Verbeken E, Lauweryns J, Flameng W. 1994. New Na+-H+ exchange inhibitor HOE 694 improves postischemic function and high-energy phosphate resynthesis and reduces Ca2+ overload in isolated perfused rabbit heart. Circulation 89:2787–2798.

4. Takeo S, Tanonaka K, Hayashi M, Yamamoto K, Liu JX, Kamiyama T, Yamaguchi N, Miura A, Natsukawa T. 1995. A possible involvement of sodium channel blockade of class-I-type antiarrhythmic agents in postischemic contractile recovery of isolated perfused hearts. J Pharmacol Exp Ther 273:1403–1409.

5. Karmazyn M. 1991. Amiloride enhances postischemic ventricular recovery: possible role of Na+/H+ exchange. Am J Physiol 255:H608–H615.

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