The Role of Viral Reactivation in Drug Reaction with Eosinophilia and Systemic Symptoms and Other Cutaneous Adverse Drug Reactions (cADRs)
Author:
Publisher
Springer Science and Business Media LLC
Subject
Dermatology
Link
http://link.springer.com/content/pdf/10.1007/s13671-016-0124-y.pdf
Reference57 articles.
1. Roujeau JC. Clinical heterogeneity of drug hypersensitivity. Toxicology. 2005;209(2):123–9.
2. Pichler WJ. Consequences of drug binding to immune receptors: Immune stimulation following pharmacological interaction with immune receptors (T-cell receptor for antigen or human leukocyte antigen) with altered peptide-human leukocyte antigen or peptide. Dermatol Sinica. 2013;31(4):181–90.
3. Takahashi R, Kano Y, Yamazaki Y, et al. Defective regulatory T cells in patients with severe drug eruptions: timing of the dysfunction is associated with the pathological phenotype and outcome. J Immunol. 2009;182(12):8071–9.
4. Chen YC, Chiang HH, Cho YT, et al. Human herpes virus reactivations and dynamic cytokine profiles in patients with cutaneous adverse drug reactions—a prospective comparative study. Allergy. 2015;70(5):568–75. This study confirmed that HHV-6 reactivation is specific for DRESS/DIHS, while EBV and CMV reactivations could be observed in various cADRs.
5. Chung WH, Hung SI, Chen YT. Human leukocyte antigens and drug hypersensitivity. Curr Opin Allergy Clin Immunol. 2007;7(4):317–23.
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