Pyoderma Gangrenosum: What Do We Know Now?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Dermatology
Link
http://link.springer.com/article/10.1007/s13671-018-0224-y/fulltext.html
Reference117 articles.
1. Scherlinger M, Guillet S, Doutre MS, Beylot-Barry M, Pham-Ledard A. Pyoderma gangrenosum with extensive pulmonary involvement. J Eur Acad Dermatol Venereol. 2017;31(4):e214–e6. https://doi.org/10.1111/jdv.13976.
2. Vadillo M, Jucgla A, Podzamczer D, Rufi G, Domingo A. Pyoderma gangrenosum with liver, spleen and bone involvement in a patient with chronic myelomonocytic leukaemia. Br J Dermatol. 1999;141(3):541–3.
3. • Wang EA, Steel A, Luxardi G, Mitra A, Patel F, Cheng MY, et al. Classic ulcerative pyoderma gangrenosum is a T cell-mediated disease targeting follicular adnexal structures: a hypothesis based on molecular and clinicopathologic studies. Front Immunol. 2017;8:1980. https://doi.org/10.3389/fimmu.2017.01980 . This study highlights the potential role of the T cell response directed against pilosebaceous units in patients with PG.
4. DeFilippis EM, Feldman SR, Huang WW. The genetics of pyoderma gangrenosum and implications for treatment: a systematic review. Br J Dermatol. 2015;172(6):1487–97. https://doi.org/10.1111/bjd.13493.
5. Antiga E, Maglie R, Volpi W, Bianchi B, Berti E, Marzano AV, et al. T helper type 1-related molecules as well as interleukin-15 are hyperexpressed in the skin lesions of patients with pyoderma gangrenosum. Clin Exp Immunol. 2017;189(3):383–91. https://doi.org/10.1111/cei.12989 .
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