Role of Ca2+-activated ion transport in the treatment of cystic fibrosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s10354-008-0596-x.pdf
Reference9 articles.
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2. Marino CR, Matovcik LM, Gorelick FS, Cohn JA. Localization of the cystic fibrosis transmembrane conductance regulator in pancreas. J Clin Invest, 88: 712–716, 1991
3. Choi JY, Muallem D, Kiselyov K, Lee MG, Thomas PJ, Muallem S. Aberrant CFTR-dependent HCO3 − transport in mutations associated with cystic fibrosis. Nature, 410: 94–97, 2001
4. Zsembery Á, Strazzabosco M, Graf J. Ca2+-activated Cl− channels can substitute for CFTR in stimulation of pancreatic duct bicarbonate secretion. FASEB J, 14: 2345–2356, 2000
5. Shumaker H, Amlal H, Frizzel R, Ulrich CD, Soleimani M. CFTR drives Na+-nHCO3 − cotransport in pancreatic duct cell: a basis for HCO3 − secretion in CF. Am J Physiol, 276: C16–C25, 1999
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2. Cigarette smoke and CFTR: implications in the pathogenesis of COPD;American Journal of Physiology-Lung Cellular and Molecular Physiology;2013-10-15
3. Proteomic and ionomic profiling reveals significant alterations of protein expression and calcium homeostasis in cystic fibrosis cells;Molecular BioSystems;2013
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