A Quinone-Containing Compound Enhances Camptothecin-Induced Apoptosis of Lung Cancer Through Modulating Endogenous ROS and ERK Signaling-Reference-Cited by-同舟云学术

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A Quinone-Containing Compound Enhances Camptothecin-Induced Apoptosis of Lung Cancer Through Modulating Endogenous ROS and ERK Signaling

Published:2016-09-27 Issue:3 Volume:65 Page:241-252
ISSN:0004-069X
Container-title:Archivum Immunologiae et Therapiae Experimentalis
language:en
Short-container-title:Arch. Immunol. Ther. Exp.

Author:

Chou Han-Lin,Fong Yao,Wei Chi-Ku,Tsai Eing-Mei,Chen Jeff Yi-Fu,Chang Wen-Tsan,Wu Chang-Yi,Huang Hurng-Wern,Chiu Chien-Chih

Funder

Ministry of Science and Technology

NSYSU-KMU Joint Research Project, Taiwan

ChiMei-KMU Joint Research Project, Taiwan

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Immunology,Immunology and Allergy

Link

http://link.springer.com/content/pdf/10.1007/s00005-016-0424-8.pdf

Reference57 articles.

1. Abrams SL, Steelman LS, Shelton JG et al (2010) The raf/mek/erk pathway can govern drug resistance, apoptosis and sensitivity to targeted therapy. Cell Cycle 9:1781–1791

2. Barrera G (2012) Oxidative stress and lipid peroxidation products in cancer progression and therapy. ISRN Oncol 2012:137289

3. Benites J, Valderrama JA, Rivera F et al (2008) Studies on quinones. Part 42: synthesis of furylquinone and hydroquinones with antiproliferative activity against human tumor cell lines. Bioorg Med Chem 16:862–868

4. Bolton JL, Trush MA, Penning TM et al (2000) Role of quinones in toxicology. Chem Res Toxicol 13:135–160

5. Brea-Calvo G, Siendones E, Sanchez-Alcazar JA et al (2009) Cell survival from chemotherapy depends on NF-kappaB transcriptional up-regulation of coenzyme q biosynthesis. PLoS One 4:e5301

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