The Potential Role of Krüppel-Like Zinc-Finger Protein Glis3 in Genetic Diseases and Cancers-Reference-Cited by-同舟云学术

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The Potential Role of Krüppel-Like Zinc-Finger Protein Glis3 in Genetic Diseases and Cancers

Published:2017-05-18 Issue:5 Volume:65 Page:381-389
ISSN:0004-069X
Container-title:Archivum Immunologiae et Therapiae Experimentalis
language:en
Short-container-title:Arch. Immunol. Ther. Exp.

Author:

Chou Chon-Kit,Tang Chin-Ju,Chou Han-Lin,Liu Chun-Yen,Ng Ming-Chong,Chang Yu-Ting,Yuan Shyng-Shiou F.,Tsai Eing-Mei,Chiu Chien-Chih^ORCID

Funder

Ministry of Science and Technology, Taiwan (TW)

Center of Excellence for Environmental Medicine, Kaohsiung Medical University (TW)

NSYSU-KMU Joint Research Project (TW)

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Immunology,Immunology and Allergy

Link

http://link.springer.com/article/10.1007/s00005-017-0470-x/fulltext.html

Reference52 articles.

1. Barthson J, Germano CM, Moore F et al (2011) Cytokines tumor necrosis factor-alpha and interferon-gamma induce pancreatic beta-cell apoptosis through STAT1-mediated Bim protein activation. J Biol Chem 286:39632–39643

2. Beak JY, Kang HS, Kim YS et al (2008) Functional analysis of the zinc finger and activation domains of Glis3 and mutant Glis3(NDH1). Nucleic Acids Res 36:1690–1702

3. Benitez CM, Goodyer WR, Kim SK (2012) Deconstructing pancreas developmental biology. Cold Spring Harb Perspect Biol 4:a012401

4. Brayer KJ, Segal DJ (2008) Keep your fingers off my DNA: protein-protein interactions mediated by C2H2 zinc finger domains. Cell Biochem Biophys 50:111–131

5. Cnop M, Welsh N, Jonas JC et al (2005) Mechanisms of pancreatic beta-cell death in type 1 and type 2 diabetes: many differences, few similarities. Diabetes 54(Suppl 2):S97–S107

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