Exposure of combination of environmental pollutant, lead (Pb) and β-amyloid peptides causes mitochondrial dysfunction and oxidative stress in human neuronal cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10863-023-09956-9.pdf
Reference55 articles.
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3. Ahmad F, Haque S, Ravinayagam V et al (2020) Developmental lead (Pb)-induced deficits in redox and bioenergetic status of cerebellar synapses are ameliorated by ascorbate supplementation. Toxicology 440:152492. https://doi.org/10.1016/j.tox.2020.152492
4. Ayyalasomayajula N, Bandaru M, Dixit PK et al (2020) Inactivation of GAP-43 due to the depletion of cellular calcium by the Pb and amyloid peptide induced toxicity: An in vitro approach. Chem Biol Interact 316:108927. https://doi.org/10.1016/j.cbi.2019.108927
5. Bandaru LJM, Ayyalasomayajula N, Murumulla L et al (2022) Defective mitophagy and induction of apoptosis by the depleted levels of PINK1 and Parkin in Pb and β-amyloid peptide induced toxicity. Toxicol Mech Methods 1–10. https://doi.org/10.1080/15376516.2022.2054749
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