miR-29b ameliorates atrial fibrosis in rats with atrial fibrillation by targeting TGFβRΙ and inhibiting the activation of Smad-2/3 pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10863-022-09934-7.pdf
Reference52 articles.
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2. Abonnenc M, Nabeebaccus AA, Mayr U, Barallobre-Barreiro J, Dong X, Cuello F et al (2013) Extracellular matrix secretion by cardiac fibroblasts: role of microRNA-29b and microRNA-30c. Circ Res 113(10):1138–1147. https://doi.org/10.1161/CIRCRESAHA.113.302400
3. Andrade J, Khairy P, Dobrev D, Nattel S (2014) The clinical profile and pathophysiology of atrial fibrillation: relationships among clinical features, epidemiology, and mechanisms. Circ Res 114(9):1453–1468. https://doi.org/10.1161/CIRCRESAHA.114.303211
4. Andrade JG, Macle L, Nattel S, Verma A, Cairns J (2017) Contemporary Atrial Fibrillation Management: A Comparison of the Current AHA/ACC/HRS, CCS, and ESC Guidelines. Can J Cardiol 33(8):965–976. https://doi.org/10.1016/j.cjca.2017.06.002
5. Bencivenga L, Komici K, Nocella P, Grieco FV, Spezzano A, Puzone B et al (2020) Atrial fibrillation in the elderly: a risk factor beyond stroke. Ageing Res Rev 61:101092. https://doi.org/10.1016/j.arr.2020.101092
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