Aberrant GSH reductase and NOX activities concur with defective CFTR to pro-oxidative imbalance in cystic fibrosis airways
Author:
Funder
Fondazione per la Ricerca sulla Fibrosi Cistica
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology
Link
http://link.springer.com/article/10.1007/s10863-018-9748-x/fulltext.html
Reference48 articles.
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2. Atlante A, Bobba A, Calissano P, Passarella S, Marra E (2003) The apoptosis/necrosis transition in cerebellar granule cells depends on the mutual relationship of the antioxidant and the proteolytic systems which regulate ROS production and cytochrome c release en route to death. J Neurochem 84:960–971. https://doi.org/10.1046/j.1471-4159.2003.01613.x
3. Atlante A, Favia M, Bobba A, Guerra L, Casavola V, Reshkin SJ (2016) Characterization of mitochondrial function in cells with impaired cystic fibrosis transmembrane conductance regulator (CFTR) function. J Bioenerg Biomembr 48:197–210. https://doi.org/10.1007/s10863-016-9663-y
4. Ballatori N et al (2009) Glutathione dysregulation and the etiology and progression of human diseases. Biol Chem 390:191–214. Review. https://doi.org/10.1515/BC.2009.033
5. Birben E, Sahiner UM, Sackesen C, Erzurum S, Kalayci O (2012) Oxidative stress and antioxidant defense. World Allergy Organ J 5:9–19. https://doi.org/10.1097/WOX.0b013e3182439613
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