TLR4 inhibitor alleviates sepsis-induced organ failure by inhibiting platelet mtROS production, autophagy, and GPIIb/IIIa expression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10863-022-09940-9.pdf
Reference38 articles.
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2. Baughman RP et al (1993) Thrombocytopenia in the intensive care unit. Chest 104(4):1243–1247
3. Bouillot S, Reboud E, Huber P (2018) Functional consequences of calcium influx promoted by bacterial pore-forming toxins. Toxins (Basel) 10(10)
4. Brennan MP et al (2009) Elucidating the role of Staphylococcus epidermidis serine-aspartate repeat protein G in platelet activation. J Thromb Haemost 7(8):1364–1372
5. Clark SR et al (2007) Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood. Nat Med 13(4):463–469
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