Protective effect of 7-oxo-prostacyclin on myocardial function and metabolism during postischemic reperfusion and calcium paradox

Author:

Ravingerová T.,Styk J.,Trégerová V.,Pancza D.,Slezák J.,Tribulová N.,Ziegelhöffer A.,Pissarek M.,Szekeres L.

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Reference16 articles.

1. Chapman RA (1987) Calcium channels, the sodium-calcium exchange and intracellular sodium in the predisposition of the heart to the calcium paradox. Biomed Biochim Acta 46:8/9, S512–516

2. Džurba A, Ziegelhöffer A, Breier A, Vrbjar N, Szekeres L (1991) Increased activity of sarcolemmal (Na+/K+)-ATPase is involved in late cardioprotective action of 7-oxo-prostacyclin on the heart. Cardioscience (in press)

3. Lamers JMJ, Ruigrok TJC (1983) Diminished Na+/K+ and Ca2+ pump activities in the Ca2+ depleted heart: possible role in the development of Ca2+ overload during the Ca2+ paradox. European Heart Journal 4 (Suppl II):73–79

4. Linz W, Lau HH, Beek G, Schölkens BH (1988) Influence of the tromboxane synthetase inhibitor HOE 944, prostacyclin and indomethacin on reperfusion arrhythmias, cardiodynamics and metabolism in isolated ischemic rat hearts. Biomed Biochim Acta 47:10/11, 23–26

5. Parratt JR, Coker SJ, Wainwright CL (1987) Eicosanoids and susceptibility to ventricular arrhythmias during myocardial ischemia and reperfusion. J Mol Cell Cardiol 19 (Suppl 5):55–66

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