Novel mitochondrial tRNAArg 10461A>G mutation in a pedigree with obesity-Reference-Cited by-同舟云学术

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Novel mitochondrial tRNAArg 10461A>G mutation in a pedigree with obesity

Published:2020-01-17 Issue:4 Volume:16 Page:429-431
ISSN:1708-8569
Container-title:World Journal of Pediatrics
language:en
Short-container-title:World J Pediatr

Author:

Wang Jin-Ling,Ji Yan-Chun^ORCID,Fu Jun-Fen

Funder

National Key R&D Program of China

Natural Science Foundation of China

Jin Lei Pediatric Endocrinology Growth Research Fund for Young Physicians

Zhejiang Provincial Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Pediatrics, Perinatology and Child Health

Link

http://link.springer.com/content/pdf/10.1007/s12519-019-00334-3.pdf

Reference17 articles.

1. Zhao NN, Dong GP, Wu W, Wang JL, Ullah R, Fu JF. FTO gene polymorphisms and obesity risk in Chinese population: a meta-analysis. World J Pediatr. 2019;15:382–9.

2. Styne DM, Arslanian SA, Connor EL, Farooqi IS, Murad MH, Silverstein JH, et al. Pediatric obesity-assessment, treatment, and prevention: an endocrine society clinical practice guideline. J Clin Endocrinol Metab. 2017;102:709–57.

3. Van der Klaauw AA, Farooqi IS. The hunger genes: pathways to obesity. Cell. 2015;161:119–32.

4. Pigeyre M, Yazdi FT, Kaur Y, Meyre D. Recent progress in genetics, epigenetics and metagenomics unveils the pathophysiology of human obesity. Clin Sci. 2016;130:943–86.

5. Li W, Wen C, Li W, Wang H, Guan X, Zhang W, et al. The tRNA(Gly) T10003C mutation in mitochondrial haplogroup M11b in a Chinese family with diabetes decreases the steady-state level of tRNA(Gly), increases aberrant reactive oxygen species production, and reduces mitochondrial membrane potential. Mol Cell Biochem. 2015;408:171–9.

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