Pathogenesis of diabetic nephropathy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
http://link.springer.com/content/pdf/10.1007/BF02480649.pdf
Reference96 articles.
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2. Kikkawa R, Arimura T, Haneda M, Nishio T, Sawada K, Yagisawa M, Shigeta Y. Current status of type 2 (non-insulin-dependent) diabetic subjects on dialysis therapy in Japan. Diabetologia 1993;36:1105–1108.
3. Low BC, Ross IK, Grigor MR: Angiotensin II stimulates glucose transport activity in cultured vascular smooth muscle cells. J Biol Chem 1992;267:20740–20745.
4. Kikkawa R, Kajiwara N, Haneda M, Shigeta Y, Tsuda H, Yano H, Seino Y. GLUT I is a main glucose transporter in rat mesangial cells. J Am Soc Nephrol 1992;3:830A.
5. Brosius III FC, Briggs JP, Marcus RG, Barac-Nieto M, Charron MJ. Insulin-responsive glucose transporter expression in renal microvessels and glomeruli. Kidney Int 1992;42:1086–1092.
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1. d-allulose protects against diabetic nephropathy progression in Otsuka Long-Evans Tokushima Fatty rats with type 2 diabetes;PLOS ONE;2022-01-31
2. Renal Function Tests on Diabetes-Induced and Non-Induced APA Hamsters.;Experimental Animals;2002
3. Response of mesangial cells to low-density lipoprotein and angiotensin II in diabetic (OLETF) rats;Kidney International;2002-01
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