Clinicopathological analysis of ANCA-associated glomerulonephritis focusing on plasma cell infiltrate

Author:

Masuzawa Naoko,Nishimura Ayako,Mihara Yu,Tamagaki Keiichi,Konishi Eiichi

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Nephrology,Physiology

Reference16 articles.

1. Brodsky S, Nadasdy T. Acute and chronic tubulointerstitial nephritis. In: Jennete JC, Olson JL, Silva FG, D’Agati VD, editors. Heptinstall’s pathology of the kidney. 7th ed. Philadelphia: Wolter Kulwer; 2015. p. 1111–1165.

2. Desvaux D, Gouvello SL, Pastural M, Abtahi M, et al. Acute renal allograft rejections with major interstitial oedema and plasma cell-rich infiltrates: high γ-interferon expression and poor clinical outcome. Nephrol Dial Transplant. 2004;19:933–9.

3. Yanagita E, Ito T. Evaluation of IgG4/IgG ratio in IgG4-related sclerosing disease using “masking double immunostaining”. Pathol Clin Med. 2010;28:987–92 (in Japanese).

4. Berden AE, Ferrario F, Hagen EC, Jayne DR, et al. Histopathologic classification of ANCA-associated glomerulonephritis. J Am Soc Nephrol. 2010;21:1628–36.

5. Tipping PG, Kitching AR. Glomerulonephritis, Th1 and Th2: What's new? Clin Exp Immunol. 2005;142:207–15.

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