Slit diaphragm dysfunction in proteinuric states: identification of novel therapeutic targets for nephrotic syndrome-Reference-Cited by-同舟云学术

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Slit diaphragm dysfunction in proteinuric states: identification of novel therapeutic targets for nephrotic syndrome

Published:2009-08 Issue:4 Volume:13 Page:275-280
ISSN:1342-1751
Container-title:Clinical and Experimental Nephrology
language:en
Short-container-title:Clin Exp Nephrol

Author:

Kawachi Hiroshi,Suzuki Koichi,Miyauchi Naoko,Hashimoto Taeko,Otaki Yasuhiro,Shimizu Fujio

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Nephrology,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s10157-009-0162-x.pdf

Reference55 articles.

1. Pavenstadt H, Kriz W, Kretzler M. Cell biology of the glomerular podocyte. Physiol Rev. 2003;83:253–307.

2. Kawachi H, Shimizu F. Molecular composition and function of the slit diaphragm: nephrin, the molecule responsible for proteinuria. Clin Exp Nephrol. 2000;4:161–72.

3. Mundel P, Shankland SJ. Podocyte biology and response to injury. J Am Soc Nephrol. 2002;13:3005–15.

4. Arakawa M. A scanning electron microscopy of the glomerulus of normal and nephrotic rats. Lab Invet. 1970;23:489–97.

5. Yamada E. The fine structure of the renal glomerulus of the mouse. J Biophys Biochem Cytol. 1955;1:551–6.

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