Altered arginine vasopressin-cyclic AMP-aquaporin 2 pathway in patients with chronic kidney disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10157-022-02220-1.pdf
Reference21 articles.
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2. Bricker NS, Dewey RR, Lubowitz H, Stokes J, Kirkensgaard T. Observations on the concentrating and diluting mechanisms of the diseased kidney. J Clin Investig. 1959;38:516–23.
3. Ettema EM, Heida J, Casteleijn NF, Boesten L, Westerhuis R, Gaillard C, et al. The effect of renal function and hemodialysis treatment on plasma vasopressin and copeptin levels. Kidney Int Rep. 2017;2:410–9.
4. Nielsen S, Kwon TH, Christensen BM, Promeneur D, Frøkiaer J, Marples D. Physiology and pathophysiology of renal aquaporins. J Am Soc Nephrol. 1999;10:647–63.
5. Imamura T, Kinugawa K, Shiga T, Kato N, Muraoka H, Minatsuki S, et al. Novel criteria of urine osmolality effectively predict response to tolvaptan in decompensated heart failure patients—association between non-responders and chronic kidney disease. Circ J. 2013;77:397–404.
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