Human MiR-4660 regulates the expression of alanine–glyoxylate aminotransferase and may be a biomarker for idiopathic oxalosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10157-019-01723-8.pdf
Reference21 articles.
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2. Salido E, Pey AL, Rodriguez R, Lorenzo V. Primary hyperoxalurias: disorders of glyoxylate detoxification. Biochim Biophys Acta. 2012;1822:1453–64.
3. Hoppe B. An update on primary hyperoxaluria. Nat Rev Nephrol. 2012;8:467–75.
4. Hoppe B, Dittlich K, Fehrenbach H, Plum G, Beck BB. Reduction of plasma oxalate levels by oral application of Oxalobacter formigenes in 2 patients with infantile oxalosis. Am J Kidney Dis. 2011;58:453–5.
5. M’Dimegh S, Aquaviva-Bourdain C, Omezzine A, M’Barek I, Souche G, Zellama D, et al. A novel mutation in the AGXT gene causing primary hyperoxaluria type I: genotype-phenotype correlation. J Genet. 2016;95:659–66.
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