The role of the ubiquitin–proteasome system in kidney diseases
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10157-012-0643-1.pdf
Reference103 articles.
1. Mitch WE, Goldberg AL. Mechanisms of muscle wasting. The role of the ubiquitin–proteasome pathway. N Engl J Med. 1996;335(25):1897–905.
2. Rajan V, Mitch WE. Ubiquitin, proteasomes and proteolytic mechanisms activated by kidney disease. Biochim Biophys Acta. 2008;1782(12):795–9. doi: 10.1016/j.bbadis.2008.07.007 .
3. Glickman MH, Ciechanover A. The ubiquitin–proteasome proteolytic pathway: destruction for the sake of construction. Physiol Rev. 2002;82(2):373–428. doi: 10.1152/physrev.00027.2001 .
4. Knecht E, Aguado C, Carcel J, Esteban I, Esteve JM, Ghislat G, et al. Intracellular protein degradation in mammalian cells: recent developments. Cell Mol Life Sci. 2009;66(15):2427–43. doi: 10.1007/s00018-009-0030-6 .
5. Reinstein E, Ciechanover A. Narrative review: protein degradation and human diseases: the ubiquitin connection. Ann Intern Med. 2006;145(9):676–84.
Cited by 20 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Renal tubular epithelial cell quality control mechanisms as therapeutic targets in renal fibrosis;Journal of Pharmaceutical Analysis;2024-01
2. Factor H-related protein 3 (FHR-3) deposition in kidney allografts – localization and correlation with complement activation;2023-10-25
3. Cytoskeletal protein degradation in brain death donor kidneys associates with adverse posttransplant outcomes;American Journal of Transplantation;2022-02-04
4. MicroRNAs in diabetic nephropathy: From molecular mechanisms to new therapeutic targets of treatment;Biochemical Pharmacology;2021-07
5. USP36-Mediated Deubiquitination of DOCK4 Contributes to the Diabetic Renal Tubular Epithelial Cell Injury via Wnt/β-Catenin Signaling Pathway;Frontiers in Cell and Developmental Biology;2021-04-23
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3