Histopathological and proteomic analyses identify integrin-β1 as a potential mediator of phlebosclerosis in uremic patients-Reference-Cited by-同舟云学术

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Histopathological and proteomic analyses identify integrin-β1 as a potential mediator of phlebosclerosis in uremic patients

Published:2019-06-18 Issue:9 Volume:23 Page:1100-1108
ISSN:1342-1751
Container-title:Clinical and Experimental Nephrology
language:en
Short-container-title:Clin Exp Nephrol

Author:

Zhou Chunyu,Li Changbin,Wang Qiang,Wu Mingyu,Mohan Chandra,Hu Dayong,Peng Ai

Funder

National Natural Science Foundation of China

Science and Technology Commission of Shanghai Municipality

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Nephrology,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s10157-019-01755-0.pdf

Reference39 articles.

1. Go AS, Chertow GM, Fan D, McCulloch CE, Hsu CY. Chronic kidney disease and the risks of death, cardiovascular events, and hospitalization. N Engl J Med. 2004;351(13):1296–305.

2. Thompson S, James M, Wiebe N, Hemmelgarn B, Manns B, Klarenbach S, et al. Cause of death in patients with reduced kidney function. J Am Soc Nephrol. 2015;26(10):2504–11.

3. Brunet P, Gondouin B, Duval-Sabatier A, Dou L, Cerini C, Dignat-George F, et al. Does uremia cause vascular dysfunction? Kidney Blood Press Res. 2011;34(4):284–90.

4. Fang Y, Ginsberg C, Sugatani T, Monier-Faugere MC, Malluche H, Hruska KA. Early chronic kidney disease-mineral bone disorder stimulates vascular calcification. Kidney Int. 2014;85(1):142–50.

5. Massy ZA, Ivanovski O, Nguyen-Khoa T, Angulo J, Szumilak D, Mothu N, et al. Uremia accelerates both atherosclerosis and arterial calcification in apolipoprotein E knockout mice. J Am Soc Nephrol. 2005;16(1):109–16.

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1. Uremic toxin indoxyl sulfate induces dysfunction of vascular smooth muscle cells via integrin-β1/ERK signaling pathway;Clinical and Experimental Nephrology;2022-03-25

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